Szybciej, lepiej, więcej mięśni. Czy właśnie tego oczekujesz po efektywnym treningu?Odchudzanie w wieku dojrzewania http: Trening w domu Z archiwum kulturystyki Targowisko Szybkie pytania bez logowania. Pierwsze badania odnosnie kreatyny w polaczeniu z treningiem - kreatyna anaboliczna ! Influence of oral creatine supplementation of muscle torque during repeated bouts of maximal testbericht odys primo exercise kreattna man. The present experiment was undertaken to investigate the influence kreatyna anaboliczna oral creatine supplementation, shown previously to increase the total creatine content of human skeletal muscle Harris RC, Soderlund K, Hultman E.
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Odchudzanie w wieku dojrzewania http: Trening w domu Z archiwum kulturystyki Targowisko Szybkie pytania bez logowania. Pierwsze badania odnosnie kreatyny w polaczeniu z treningiem - rok ! Influence of oral creatine supplementation of muscle torque during repeated bouts of maximal voluntary exercise in man.
The present experiment was undertaken to investigate the influence of oral creatine supplementation, shown previously to increase the total creatine content of human skeletal muscle Harris RC, Soderlund K, Hultman E. Clin Sci ; Muscle torque production and plasma ammonia and blood lactate accumulation were measured during and after exercise on each treatment. No difference was seen when comparing muscle peak torque production during exercise before and after placebo ingestion.
Plasma ammonia accumulation was lower during and after exercise after creatine ingestion. No differences were found when comparing blood lactate levels. Suplementacja kreatyna jest bardziej 'skuteczniejsza' i efektywniejsza u osob z naturalnie mniejszymi zasobami kreatyny: Elevation of creatine in resting and exercised muscle of normal subjects by creatine supplementation.
Supplementation with 5g of creatine monohydrate, four or six times a day for 2 or more days resulted in a significant increase in the total creatine content of the quadriceps femoris muscle measured in 17 subjects. This was greatest in subjects with a low initial total creatine content and the effect was to raise the content in these subjects closer to the upper limit of the normal range. Cr ingestion produced a Cumulative loss of ATP was Changes in PCr before exercise bouts 1 and 2 in type II fibers were positively correlated with changes in PCr degradation during exercise in this fiber type and changes in total work production.
The results suggest that improvements in performance were mediated via improved ATP resynthesis as a consequence of increased PCr availability in type II fibers. This study was undertaken to investigate the influence of oral supplementation with creatine monohydrate on muscular performance during repeated sets of high-intensity resistance exercise.
Before T1, both groups received no supplementation. From T1 to T2, both groups ingested placebo capsules. From T2 to T3, the creatine group ingested 25 g creatine monohydrate per day, and the placebo group ingested an equivalent amount of placebo. Total repetitions for each set of bench presses and peak power output for each set of jump squats were determined. Other measures included assessment of diet, body mass, skinfold thickness, and preexercise and 5-minute postexercise lactate concentrations.
Lifting performance was not altered for either exercise protocol after ingestion of the placebos. Creatine supplementation resulted in a significant improvement in peak power output during all 5 sets of jump squats and a significant improvement in repetitions during all 5 sets of bench presses. After creatine supplementation, postexercise lactate concentrations were significantly higher after the bench press but not the jump squat. A significant increase in body mass of 1.
Effects of creatine on isometric bench-press performance in resistance-trained humans. The purpose of this study was to investigate the effects of creatine Cr supplementation on force generation during an isometric bench-press in resistance-trained men. Subjects performed an isometric bench-press test involving five maximal isometric contractions before and after 5 d of Cr 20 g. Body composition was measured before and after supplementation. Subjects completed h urine collections throughout the study period; these were subsequently analyzed to provide total Cr and creatinine excretion.
For the Cr group, peak force and total force pre- or post-supplementation were not different from placebo. However, when the analysis was confined to the responders, both the change in peak force [Repetition 2: Cr significantly increased body weight Five days of Cr supplementation increased body weight and fat-free body mass in resistance-trained men who were classified as responders.
Peak force and total force during a repeated maximal isometric bench-press test were also significantly greater in the responders compared to the placebo group http: Combined creatine and protein supplementation in conjunction with resistance training promotes muscle GLUT-4 content and glucose tolerance in humans.
The present study was undertaken to explore the effects of creatine and creatine plus protein supplementation on GLUT-4 and glycogen content of human skeletal muscle.
This was investigated in muscles undergoing a decrease immobilization and subsequent increase resistance training in activity level, compared with muscles with unaltered activity pattern.
A double-blind, placebo-controlled trial was performed by 33 young healthy subjects. The subjects' right legs were immobilized with a cast for 2 wk, followed by a 6-wk resistance training program for the right knee extensor muscles.
Needle biopsies were bilaterally taken from the vastus lateralis. Supplements had no effect on GLUT-4 expression or glycogen content in contralateral control legs.
We conclude that creatine intake stimulates GLUT-4 and glycogen content in human muscle only when combined with changes in habitual activity level. Furthermore, combined protein and creatine supplementation improved oral glucose tolerance, which is supposedly unrelated to the changes in muscle GLUT-4 expression. Stosowanie kreatyny lub kreatyny wraz z bialkiem przynosi korzystne efekty tylko! Efects of creatine supplementation and resistance training on muscle strength and weightlifting performance.
Creatine monohydrate has become the supplement of choice for many athletes striving to improve sports performance. Recent data indicate that athletes may not be using creatine as a sports performance booster per se but instead use creatine chronically as a training aid to augment intense resistance training workouts.
Although several studies have evaluated the combined effects of creatine supplementation and resistance training on muscle strength and weightlifting performance, these data have not been analyzed collectively. The purpose of this review is to evaluate the effects of creatine supplementation on muscle strength and weightlifting performance when ingested concomitant with resistance training.
The effects of gender, interindividual variability, training status, and possible mechanisms of action are discussed. Thus there is substantial evidence to indicate that creatine supplementation during resistance training is more effective at increasing muscle strength and weightlifting performance than resistance training alone, although the response is highly variable. Athletic performance enhancement enhanced muscle mass and muscle strength Several high-quality studies have shown an increase in muscle mass with the use of creatine.
However, some weaker studies have reported mixed results. Overall, the available evidence suggests that creatine does increase lean body mass, strength, and total work.
Future studies should take into account the effect of different individual fitness levels of study subjects. Congestive heart failure chronic Patients with chronic heart failure have low levels of creatine in their hearts. Several studies report that creatine supplements may improve heart muscle strength, body weight, and endurance in patients with heart failure. Studies comparing creatine with drugs used to treat heart failure are needed before a firm recommendation can be made.
Heart failure should be treated by a qualified healthcare professional. Creatine can be synthesized in the human body from dietary amino acids. Synthesis begins in the kidney when arginine and glycine form guanidoacetic acid. This product is methylated in the liver, forming creatine, technically known as methylguanidine-acetic acid.
This decrease in circulating guanidinoacetate levels suggests that exogenous supply of creatine chronically inhibits endogenous synthesis at the transamidinase step in humans. This may lead to enhanced utilization of arginine as a substrate for secondary pathways. Creatine kinase transfers the phosphate group from adenosine triphosphate ATP to creatine, thereby forming phosphocreatine plus adenosine diphosphate ADP.
This process is reversible in muscle cells and maintains the cellular ATP: Creatine kinase is high in cells undergoing high-energy fluctuations, such as muscle cells, cardiac muscle cells, neurons, photoreceptor cells, and spermatozoa. Creatine supplementation has a greater effect on phosphocreatine availability and resynthesis rate in middle-aged compared with younger persons. Aging is associated with lower total creatine and phosphocreatine concentrations and loading may be more efficient in subjects with a lower basal creatine level.
To date creatine-induced effects of increased muscle strength, body mass, and performance have not been confirmed in studies on elderly subjects. Creatine, like antioxidants, may upregulate apoptosis in preneoplastic and neoplastic cells. In vitro data suggest that cyclocreatine, a creatine kinase substrate analog, is cytotoxic to many solid tumors including human ME cervical carcinoma. Addition of creatine and phosphocreatine analogs to the MCF-7 breast adenocarcinoma and the HT colon adenocarcinoma cell lines delay the growth of rat mammary adenocarcinomas comparable to effects seen with currently used anticancer drugs.
Nude mice transplanted with human colon adenocarcinoma cells given creatine demonstrated significant inhibition of tumor growth; the growth inhibition was directly correlated with creatine tissue concentrations.
Cyclocreatine, an analog of creatine, was effective when added to standard anticancer agents including cis-diaminedichloroplatinum II , cyclophosphamide, adriamycin, or 5-fluorouracil, and resulted in tumor-growth delay in SW2 small-cell lung cancer cells as compared with those obtained for each of the drugs alone and when administered to rats with mammary carcinoma. Myocytes use creatine to make phosphocreatine PCr via the enzyme creatine kinase CK.
PCr also buffers intracellular hydrogen ions associated with lactate production and muscle fatigue during exercise. Thus, creatine may increase both the force of muscle contraction by boosting ATP levels and the duration of anaerobic exercise. During the first 10 seconds of intense exercise, creatine levels are markedly depleted. Creatine supplementation may accelerate PCr resynthesis following intense muscle contractions. Creatine supplementation has been shown to increase muscle creatine levels.
In clinical trials, creatine has increased body mass. An increase in intracellular water has been suggested as a mechanism for increasing body mass. In one study examining the effect of creatine in the elderly, it was determined that creatine may increase bone density in combination with resistance training. The mechanism for this effect is unclear.
However, in another clinical trial, creatine had no effect on these lipid parameters. Exercise-induced myocardial ischemia is accompanied by increased serum creatine concentrations. No effect was noted on homocysteine levels in healthy individuals. Various clinical studies, case series, and randomized trials support the use of creatine supplementation for congestive heart failure. In a controlled study, no changes were observed in those taking HMB plus creatine for six weeks. Decreased glucose levels in creatine-supplemented animals have been suggested.
In humans, neither acute nor short-term creatine supplementation influenced glucose tolerance or levels. High concentrations of insulin may enhance muscle creatine accumulation. This is a result of an insulin-induced transport of creatine from circulation to skeletal muscle vs.
Decreased insulin levels in animals have been suggested.